Researcher.Life Logo

Journal of Neuroinflammation : Impact Factor & More

eISSN: 1742-2094pISSN: 1742-2094
JournalOpen Access

Aims and Scope of Journal of Neuroinflammation

The Journal of Neuroinflammation is a peer-reviewed open-access scientific journal covering immunological responses of the nervous system. It was established in 2004 and is published by BioMed Central. The editors-in-chief are Sue T. Griffin (University of Arkansas for Medical Sciences) and Monica J. Carson (University of California, Riverside), who succeeded Robert E. Mrak (University of Toledo Medical Center) in 2018. Less

Key Metrics

CiteScore
13.4
Eigenfactor
0.01 - 0.05
Impact Factor
5 - 10
Scite Index
0.92 5-Year SI
SJR
Q1Neurology
SNIP
1.81
Recommended pre-submission checks
Powered by Paperpal by Editage

Topics Covered on Journal of Neuroinflammation

Journal of Neuroinflammation Journal Specifications

Indexed in the following public directories

  • Web of Science Web of Science
  • Scopus Scopus
  • DOAJ DOAJ
  • SJR SJR
Overview
Publisher BMC
Language English
Frequency Continuous publication
Article Processing ChargesEUR 2890 | USD 3490 | GBP 2490
Publication Time12
Editorial Review ProcessAnonymous peer review
General Details
LanguageEnglish
FrequencyContinuous publication
Publication Start Year2004
Publisher URLVisit website
Website URLVisit website
Publication Details
PlagiarismVisit website
Publication Time 12
Waiver PolicyVisit website
Editorial Review Detail
Editorial TeamVisit website
Review ProcessAnonymous peer review
Review UrlVisit website
Information for authors
Author instructionsVisit website
Copyright DetailsVisit website
Deposit PolicySherpa/Romeo
License typeCC BY, CC0
OA statementVisit website
View less

Planning to publish in Journal of Neuroinflammation ?

Upload your Manuscript to get

  • Degree of match
  • Common matching concepts
  • Additional journal recommendations
Free Report

Recently Published Papers in Journal of Neuroinflammation

NAD+ depletion drives age-related monocyte hyperinflammation after stroke and is reversed by nicotinamide riboside.
  • 27 Nov 2025
  • Journal of neuroinflammation
Granzyme B contributes to subretinal fibrosis in neovascular age-related macular degeneration by modulating inflammation and epithelial-mesenchymal transition
  • 27 Nov 2025
  • Journal of Neuroinflammation
Post-onset intermittent fasting attenuates neuroinflammation and demyelination via a TRIB3-PERK-autophagy axis in an EAE model of multiple sclerosis.
  • 27 Nov 2025
  • Journal of neuroinflammation
Inhibition of bruton’s tyrosine kinase directly alleviates pathogenic CD4+ T cell response via PPAR-γ signaling in autoimmune uveitis
  • 27 Nov 2025
  • Journal of Neuroinflammation
FOXA2 regulated by HDAC3-mediated deacetylation attenuates neuropathic pain by modulating microglial lipid metabolism and synaptic pruning dysregulation
  • 26 Nov 2025
  • Journal of Neuroinflammation
Metabolic reprogramming and altered ATP content impair neuroprotective functions of microglia in β-glucocerebrosidase deficiency models.
  • 25 Nov 2025
  • Journal of neuroinflammation
NAD+ depletion drives age-related monocyte hyperinflammation after stroke and is reversed by nicotinamide riboside.
  • 27 Nov 2025
  • Journal of neuroinflammation
Granzyme B contributes to subretinal fibrosis in neovascular age-related macular degeneration by modulating inflammation and epithelial-mesenchymal transition
  • 27 Nov 2025
  • Journal of Neuroinflammation
Post-onset intermittent fasting attenuates neuroinflammation and demyelination via a TRIB3-PERK-autophagy axis in an EAE model of multiple sclerosis.
  • 27 Nov 2025
  • Journal of neuroinflammation
Inhibition of bruton’s tyrosine kinase directly alleviates pathogenic CD4+ T cell response via PPAR-γ signaling in autoimmune uveitis
  • 27 Nov 2025
  • Journal of Neuroinflammation
FOXA2 regulated by HDAC3-mediated deacetylation attenuates neuropathic pain by modulating microglial lipid metabolism and synaptic pruning dysregulation
  • 26 Nov 2025
  • Journal of Neuroinflammation
Metabolic reprogramming and altered ATP content impair neuroprotective functions of microglia in β-glucocerebrosidase deficiency models.
  • 25 Nov 2025
  • Journal of neuroinflammation

FAQs on Journal of Neuroinflammation